Targeted Therapy in Acute Myeloid Leukemia: Current Approaches and Novel Directions.

Publication Type Review
Authors Ko K, Gelfer R, Wheat J, Cai S
Journal J Pers Med
Volume 16
Issue 3
Date Published 03/20/2026
ISSN 2075-4426
Abstract Acute myeloid leukemia (AML) is a molecularly heterogeneous neoplasm of hematopoietic stem and progenitor cells. The advent of high-resolution genomic sequencing has uncovered several genetic drivers of AML which spurred a surge of therapies that target the disease at a mutational, clonal, or epigenetic level. Currently, the molecular profiling of AML patients before treatment is commonplace and crucial for ensuring that patients receive the most optimal therapy for any driver mutations they may have. Here, we detail the current targeted therapies available for AML: specifically, those targeting the BCL2 family (venetoclax), FLT3 (midostaurin, gilteritinib, quizartinib), IDH1/2 (enasidenib, ivosidenib), and MENIN (revumenib, ziftomenib). In addition, we outline potential mechanisms of resistance against these therapies, as well as efforts being taken to prevent or bypass them.
DOI 10.3390/jpm16030169
PubMed ID 41893037
PubMed Central ID PMC13028391
Back to Top